Nicholas K. Gonatas

Nicholas K. Gonatas is Professor Pathology and Laboratory Medicine at the University of Pennsylvania, Philadephia.

• D. L. Gasser, J. Palm, and N. K. Gonatas:
  Genetic control of susceptibility to experimental allergic encephalomyelitis and the Ag-B locus of rats.
  Journal of Immunology, Baltimore, 1975: 115: 431-433.
• D. L. Gasser, S. J. Baloyannis, and N. K. Gonatas.
  Influence of MHC-linked and non-MHC-linked genes on susceptibility of rats to experimental allergic encephalomyelitits. In N. R. Rose, P. E. Bigazzi, and N. L. Warner, eds., Genetic Control of Autoimmune Disease, 355364, Elsevier-North Holland, New York, 1978.
• R. J. Packer, D. R. Cornblath, N. K. Gonatas, L. A. Bruno, A. K. Asbury:
  Creutzfeldt-Jacob disease in a 20-year old woman.
  Neurology, 1980, 30: 492-496.
• J. W. Trojanowski, J.O. Gonatas, and N.K. Gonatas:
  Horseradish peroxidase conjugates of cholera toxin and lectins are more sensitive retrogradely transported markers than free HRP.
  Brain Research, Amsterdam, 1982, 231: 33-50.
• D. L. Gasser, W. F. Hickey, and N. K. Gonatas:
  The genes for nonsusceptibility to EAE in the Le-R and BH rat strains are not linked to RT1.
  Immunogenetics, New York, 1983, 17: 441-444.
• Jacqueline O. Gonatas, Zissimos Mourelatos, Anna Stieber, William S. Lane, Jurgen Brosius, Nicholas K. Gonatas:
  MG-160, a membrane sialoglycoprotein of the medial cisternae of the rat Golgi apparatus, binds basic fibroblast growth factor and exhibits a high level of sequence identity to a chicken fibroblast growth factor receptor.
  Journal of Cell Science, Cambridge, 1995, 108 (2): 457-467.
• J. O. Gonatas, Y.-J. Chen, A. Stieber, Z. Mourelatos, and N. K. Gonatas:
  Truncations of the C-terminal cytoplasmic domain of MG160, a medial Golgi sialoglycoprotein, result in its partial transport to the plasma membrane and filopodia.
  Journal of Cell Science, Cambridge, 1998, 111: 249-260.
• Stieber, Y.-J. Chen, S. Wei, Z. Mourelatos, J. Gonatas, and N. K. Gonatas:
  The fragmented neuronal Golgi apparatus in amyotrophic lateral sclerosis includes the trans-Golgi-netwotk: functional implications.
  Acta neuropathologica, Berlin, 1998, 95: 245-253.
• N. K. Gonatas, J. Gonatas, and A. Stieber:
  The involvementof the Golgi apparatus in the pathogenesis of amyotrophic lateralsclerosis, Alzheimer's disease and ricin intoxication.
  Histochemistry and Cell Biology, Berlin, 1998, 109: 591-600.
• A.-H. Monsoro-Burq, A. Stieber, M. Bontoux, N. Le Douarin, and N. K. Gonatas:
  Environmental factors modulate the size and thesecretory activity of the notochord. A study og the Golgi apparatus in avian embryos.
  Comptes rendus de l'Académie des Sciences. Série III : Sciences de la vie/Life Sciences, Paris, 1998, 321: 621-631.
• Y. Fujita, K. Okamoto, A. Sakurai, M. Amati, Y. Nakazano, and N. K. Gonatas:
  Fragmentation of the Golgi apparatus of Betz cells in patients with amyotrophic lateral sclerosis.
  Journal of Neurological Science, 1999, 163: 81-85.
• Salehi, C.W. Pool, M. Mulder, R. Ravid, N.K. Gonatas and D.F. Swaab:
  Activity of hippocampal CA1 neurons in Alzheimer's disease is not affected by the presence of adjacent neuritic plaques.
  JAD Journal of Alzheimer's Disease, 1998, 1 (2): 107-118.
• Anna Stieber, Youjun Chen, Shuang Wei, Zissimos Mourelatos, Jacqueline Gonatas, Koichi Okamoto, N. K. Gonatas:
  The fragmented neuronal Golgi apparatus in amyotrophic lateral sclerosis includes the trans-Golgi-network: functional implications.
  Acta neuropathologica, Berlin, 1998, 95, 3: 245-253.
• Sakurai, K. Okamoto, Y. Fujita, Y. Nakazato, K. Wakabayashi, H. Takahashi, N. K. Gonatas:
  Fragmentation of the Golgi apparatus of the ballooned neurons in patients with corticobasal degeneration and Creutzfeldt-Jakob disease.
  Acta neuropathologica, Berlin, 2000, 100, 3: 270-274
• N. K. Gonatas:
  The Golgi apparatus in disease. In: The Golgi Apparatus.
  E.G. Berger, University of Zürich, editor, Switzerland, J. Roth, University of Zürich, editor. 1997.